Ear Pain
Introduction
Ear pain (otalgia) is a common presenting complaint confronting the practicing GMO.
Pain may be caused by intrinsic otopathology or might be referred from a distant
pathologic process. Even though the most common cause of otalgia is infection, anything
from a foreign body to barotrauma to oropharyngeal tumors may cause this symptom. Your
approach should focus on the diagnosis and treatment of acute processes while excluding
serious pathology.
History
Obtain a detailed history. Inquire about symptoms of pain, discharge, hearing loss,
vertigo, and pain in other areas such as the pharynx or larynx. Conversely, an
oropharyngeal process may cause "referred" ear pain. Severe otalgia may radiate
to the jaw or neck. Determine if there was a precedent upper respiratory infection before
the onset of otalgia that might suggest an infectious process such as acute sinusitis or
otitis media. Ask about swimming history (swimmers ear) or exposure to rapid pressure
changes (usually on descent) from flying or deep sea diving. This may indicate barotrauma
complicated by oxygen otitis media. Constitutional symptoms may include fever, chills,
positional vertigo, tinnitus, nausea and vomiting, sore throat, dental pain, headache, and
cranial nerve palsies. A review of symptoms must include respiratory, neurologic,
metabolic, cardiovascular, and infectious complaints. Review the past medical history for
any similar symptoms and for any chronic medical conditions that may predispose to this
complaint.
Physical exam
The primary focus is on the ear, oropharynx, and nasopharynx (i.e.,
eustachian tube). Also perform a neck exam and a brief neurologic exam with attention to
gait, cranial nerves, and the presence or absence of positional nystagmus. Examination of
the ear begins with several simple clinical tests to evaluate the presence or absence of
hearing loss. Hearing loss is divided into conductive and sensorineural.
-
Lesions in the sound conducting system, which includes the ear canal and drum, middle
ear, and ossicles, cause a conductive loss.
-
Sensorineural loss is secondary to abnormalities of the vestibulocochlear system.
Begin with the voice test by occluding one ear while whispering or rubbing fingers
together and repeat for the other side. Hearing is likely normal if the patient can pass
these tests. Rinne's test evaluates conductive hearing loss by use of a 512 Hz
tuning fork. The tuning fork is struck, placed on the mastoid process, and then 1 to 2
inches away from the ear. The patient is asked which was louder. A normal (positive
Rinne's) test is when air conduction is greater than bone conduction (air conduction (AC)
> bone conduction (BC)). Remember that this test by itself is unreliable if the patient
has total neural deafness in one ear. This will be differentiated with the Weber's test.
After striking the tuning fork, place it on the midline of the skull (forehead or teeth).
Ask the patient where the sound is loudest. A normal test is when sound is heard in the
middle of the head. Unilateral conductive loss will present with sound louder in the
abnormal ear. With a sensorineural hearing loss, the sound is heard in the normal ear.
The external ear including the periauricular areas (mastoid and lymph nodes), pinna,
and external canal area are examined for evidence of trauma (laceration and hematoma) and
infection (erythema, drainage, or pain with movement of the pinna). The tympanic membranes
need to be examined for evidence of perforation (common for barotrauma and Q-tip usage),
erythema, normal landmarks (retraction or bulging), mobility, bullae, and effusions
(serous, suppurative, or bloody). Examination of the oropharynx and surrounding structures
is necessary to exclude sources of referred pain. Malocclusions and otalgia suggest TMJ
syndrome. Assess for evidence of sinusitis by palpating for facial tenderness. Examine the
oropharynx for dental lesions (tooth decay, gingivitis, abscess), peritonsillar abscess,
or tumors.
Differential Diagnosis of Otalgia
Determine whether the pain is localized or referred.
-
Local: trauma, either direct or indirect (hematoma, barotrauma, perforation), external
otitis, furuncle, otitis media (acute or chronic).
-
Referred: sinusitis, TMJ syndrome, dental disease, peritonsillar abscess, cervical
arthritis, occipital neuralgia, foreign bodies of the hypopharynx, larynx, cervical
esophagus, and less commonly, lesions involving cranial nerves VII, IX, and X such as
disease of the thyroid, chest, and abdomen. Herpes Zoster oticus and Bell's palsy may also
produce referred pain with cranial nerves V and VII.
General Treatment Guidelines
Generally treat the underlying condition.
This condition is most commonly caused by Pseudomonas and less commonly by fungi.
Treatment includes thorough suctioning and placement of a cotton wick saturated with
Cortisporin Otic suspension for 5 to 7 days. Domboro's Otic solution is useful as an
astringent and maintaining an acidic pH. Advise the patient to keep the ear canal dry.
Remember that this condition is painful and will usually require a strong analgesic.
-
Bullous Myringitis
Causes can include Mycoplasma or viral infections. If you suspect a bacterial
cause, prescribe a 10 day course of erythromycin or tetracycline.
-
Otitis Media
This condition is most commonly caused by Streptococcus pneumoniae followed by H.
influenzae, and then Moraxella. Less frequent bacterial pathogens associated with otitis
media include Streptococcus pyogenes and Staphlococcus aureus. Amoxacillin, followed by
Augmentin or Pediazole, are the first line drugs of choice. Second or third-line agents
for persistent infection include second or third generation cephalosporins and the
macrolides including Biaxin and Zithromax. Other effective antibiotics are Pediazole and Septra. Myringotomy is indicated in patients with severe pain, toxicity, persistent high
fever, or with associated complications such as facial nerve palsies, meningitis, or brain
abscess.
-
Barotitis
Inner ear inflammation caused by exposure to differing atmospheric pressures should be
treated with analgesics and oral decongestants. Antibiotics are rarely indicated.
-
TM Perforations
These injuries generally heal spontaneously without further treatment. Patients should
be instructed to avoid water in the ear. If the ear is contaminated with saltwater,
swimming pool water, or by a penetrating object, the patient should be placed on a course
of systemic antibiotics such as Dicloxacillin or Augmentin. Large perforations will
require urgent ENT referral.
The majority of patients with otalgia can be managed effectively by these simple
treatment guidelines and will not require referral.
Indications for Immediate Referral to ENT
-
Acute otitis media or sinusitis complicated by meningitis, cranial nerve involvement,
focal neurologic deficits, labyrinthitis, mastoiditis, and cavernous sinus thrombosis.
-
Gradenigo's syndrome (chronically draining ear with ipsilateral abducens palsy and pain
behind the ipsilateral orbit).
-
Systemic toxicity.
-
Peritonsillar abscess.
-
Retropharyngeal abscess.
-
Neck abscess.
-
Dental abscess (dental referral).
-
Spreading periauricular cellulitis unresponsive to antibiotics.
-
Tumors.
Non-emergent referrals would include:
-
Chronic sinusitis
-
Chronic otitis externa and media.
-
TMJ unresponsive to conservative measures (dental referral).
-
Recurrent vertigo.
-
Non-healing or large tympanic membrane perforations.
-
Recurrent tonsillitis.
-
Nasal or sinus polyps (especially if on flight status).
References
-
Karmody, C. Textbook of Otolaryngology. Lea & Febiger, 1983
-
Rosen, P. Emergency Medicine, Concepts and Clinical Practice. Second edition, C.V. Mosby
Co., 1988.
-
Tintinalli, J. Emergency Medicine, Comprehensive Study Guide. McGraw Hill, 4th
Edition, 1996.
-
Fairbanks, D. Antimicrobial Therapy in Otolaryngology-Head and Neck Surgery. American
-
Academy of Otolaryngology-Head and Neck Foundation Inc. 5th Edition, 1989
Revised by LCDR Kerry J. King, MC, USN, Emergency Department, Naval Medical
Center, San Diego, CA. Reviewed by CAPT David H. Thompson, Department of Otolaryngology,
National Naval Medical Center, Bethesda, MD. (1999).
|
Preface
· Administrative Section
· Clinical Section
The
General Medical Officer Manual , NAVMEDPUB 5134, January 1, 2000
Bureau
of Medicine and Surgery, Department of the Navy, 2300 E Street NW, Washington, D.C.,
20372-5300
This
web version of The General Medical Officer Manual, NAVMEDPUB 5134 is provided by
The Brookside Associates
Medical Education Division. It contains original contents from the
official US Navy version, but has been reformatted for web access and
includes advertising and links that were not present in the original version.
This web version has not been approved by the Department of the Navy or the
Department of Defense. The presence of any advertising on these pages does not
constitute an endorsement of that product or service by either the Department of
Defense or the Brookside Associates. The Brookside Associates is a private
organization, not affiliated with the United States Department of Defense. All
material in this version is unclassified. This formatting © 2006
Medical Education Division,
Brookside Associates, Ltd.
All rights reserved.
Home
·
Textbooks and Manuals
·
Videos
·
Lectures
·
Distance Learning
·
Training
·
Operational Safety
·
Search
This website is dedicated to the development and dissemination of medical information that may be useful to those who practice Operational Medicine. This website is privately-held and not connected to any governmental agency. The views expressed here are those of the authors, and unless otherwise noted, do not necessarily reflect the views of
the Brookside Associates, Ltd., any governmental or private organizations. All writings, discussions, and publications on this website are unclassified.
© 2006 Medical Education Division, Brookside Associates, Ltd. All rights reserved
Other Brookside Products
Contact Us
|
|